Lynn Schnapp, MD
Madison WI 53705-2281
Dr. Lynn M. Schnapp is the George R. and Elaine Love Professor and chair of the Department of Medicine. She is a faculty member in the Division of Allergy, Pulmonary and Critical Care Medicine and a graduate fellow of the Hedwig van Ameringen Executive Leadership in Academic Medicine Program. Previously, she was the Division Chief of Pulmonary, Critical Care, Allergy and Sleep Medicine at the Medical University of South Carolina (MUSC), and held faculty positions at University of Washington, Icahn School of Medicine at Mount Sinai, and University of California, San Francisco.
Dr. Schnapp is a fellow and past president of the American Thoracic Society, where she has held numerous leadership positions, and a recipient of its Elizabeth Rich Award for outstanding scientific, leadership and mentorship contributions in pulmonary medicine. She is a member of the Alpha Omega Alpha Honor Medical Society and the National Institutes of Health/National Heart, Lung and Blood Institute Advisory Council (NIH/NHLBI).
She is recognized for her long-standing commitment to mentorship, career development and increasing the pipeline of physician-scientists in medicine. She has been an active mentor in such programs as the National Institutes of Health (NIH) Maximizing Access for Research Careers (MARC) Program and the NIH/NHLBI Stipends for Training Aspiring Researchers (STAR) Program.
Dr. Schnapp is an established NIH-funded investigator in cell and molecular biology with clinical and research expertise in lung injury and repair. Her research examines matrix remodeling in the normal and injured lung and the role of lung pericytes and other stromal cells in the pathogenesis of fibrosis.
Dr. Schnapp’s clinical interests include acute respiratory distress syndrome (ARDS) and critical care medicine.
View Dr. Lynn M. Schnapp’s publications on NCBI My Bibliography
Dr. Schnapp’s research broadly focuses on the processes that govern acute lung injury and its resolution. In particular, she is interested in why lung injury resolves under certain circumstances, such as ARDS and pneumonia, but progresses to end-stage damage or fibrosis in other circumstances, such as emphysema or idiopathic pulmonary fibrosis.
To examine these questions, she uses different mouse models of lung injury to examine the regulation of matrix remodeling and the role of the alveolar myofibroblasts and lung pericytes in the resolution of injury and fibrosis.
To complement these studies, she analyzes bronchoalveolar lavage fluid and peripheral blood from patients with ARDS and other lung diseases using cutting-edge methodologies such as transcriptomics and proteomics to identify new biomarkers, pathways and molecular targets.